Asthma Case Study Example | Topics and Well Written Essays - 1250 words

Asthma - Case Study ExampleAn asthma exacerbation fag end be performd by allergens (Sequeria & Steward, 2007). There are those who only have asthma attacks as a leave alone of an allergic reaction. firearm it still remains unclear just what causes asthma in general, every soulfulness varies as to what their asthma triggers are. Asthma a condition that results from three major processes, which are airline inflammation, intermittent air flow obstruction, and bronchial hyperresponsiveness (Morris & Mosenifar, 2011)..These three processes are present in every episode of asthma respiratory tract Inflammation Airway inflammation is the result of inhaling substances that are triggers for the patient (Sequeria & Steward, 2007). Substances such as toxins that are inhaled base activate airway mast cells which bear IgE antibodies. The antibodies lead to inflammatory responses as the mediators, such as histamine, cause mucus hypersecretion and plasma leakage (Sequeria & Steward, 2007).T his inflammation results in epithelial changes, basement membrane changes, and submucosal infiltration with activated lymphocytes and eosinophils (Guill, 2004). Chronic airway inflammation drop result in airway reconstruction and make upd airway resistance (Sequeira & Stewart, 2007). Intermittent airflow Obstruction The mucus hypersecretion that is a result of the airway inflammation causes airflow obstruction. This obstruction makes it very difficult for the patient to breathe, as fresh air cannot be received by the lungs. It is also often the cause of the coughing that often coiffes as one of the symptoms of asthma. Airflow obstruction is considered to be often reversible, unlike the restructuring that may result from chronic airway inflammation (Sequeira & Stewart, 2007). However, such obstruction has render the concern that earlier and more aggressive interventions may be necessary (Guill, 2004). Airflow obstruction can become very severe and even fatal. Without any interve ntion the hypersecretion of mucus can fill the lungs and occlusive the ability to breathe altogether. Bronchial Hyperresponsiveness There is a lack of complete understanding when it comes to bronchial hyperresponsiveness (OConnor, 1993). While the understanding of this process in not complete, there have been some additions to the knowledge base brought slightly by numerous clinical studies. The search to fully understand the process persists. What is known about bronchial hyperresponsiveness in asthma is that it is directly correlated with disease severity (OConnor, 1993). There is a relationship amidst the airway inflammations that is characteristic in asthma to bronchial hyperresponsiveness, but, unlike inflammation, bronchial hyperresponsiveness can not serve as an asthma diagnostic. Instead, bronchial hyperresponsiveness has been defined as a functional disorder reflecting a movement to airflow obstruction (OConnor, 1993). Bronchial Hyperresponsiveness only serves to compou nd the problem of airflow limitations, as it works to increase dyspena. The bronchodilators that are often used in the sermon of asthma have no benefits in the treatment of bronchial hyperresponsiveness. (Van Schayck & Van Herwaarden, 1993). In addition to inhaled toxic substances as triggers, it has been found that asthma can be the result of nerve activity in some patients. There are